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Mutlu, Gokhan M.
One or more keywords matched the following properties of Mutlu, Gokhan M.
PropertyValue
keywords Acute Lung Injury
overview Dr. Mutlu is Professor of Medicine and Chief of the Section of Pulmonary and Critical Care Medicine at the University of Chicago. He is also the principal investigator/director of the NIH T32 Research training in respiratory biology. Dr. Mutlu's research focuses on the mechanisms of acute lung injury, and pulmonary fibrosis. Specifically, his laboratory is interested in understanding the role of macrophage and fibroblast metabolism including mitochondria in the pathogenesis of acute lung injury and pulmonary fibrosis. Dr. Mutlu's laboratory has extensive expertise with in vitro cell based methods including bioenergetics and in vivo models of acute lung injury (influenza, LPS, bacterial pneumonia and particulate matter) and pulmonary fibrosis (bleomycin).
One or more keywords matched the following items that are connected to Mutlu, Gokhan M.
Item TypeName
Academic Article Epithelial cell death is an important contributor to oxidant-mediated acute lung injury.
Academic Article Leptin resistance protects mice from hyperoxia-induced acute lung injury.
Academic Article Intratracheal administration of influenza virus is superior to intranasal administration as a model of acute lung injury.
Academic Article Incidence and outcomes of acute lung injury.
Academic Article Acute lung injury does not impair adenoviral-mediated gene transfer to the alveolar epithelium.
Academic Article Suppression of inflammation and acute lung injury by Miz1 via repression of C/EBP-d.
Academic Article Keratinocyte growth factor expression is suppressed in early acute lung injury/acute respiratory distress syndrome by smad and c-Abl pathways.
Academic Article Predictors of acute lung injury and severe hypoxemia in patients undergoing operative talc pleurodesis.
Academic Article Reply: ß2-agonists and acute respiratory distress syndrome.
Academic Article Alcohol worsens acute lung injury by inhibiting alveolar sodium transport through the adenosine A1 receptor.
Academic Article ß2-agonists and acute respiratory distress syndrome.
Academic Article Potential genetic therapies for acute lung injury.
Academic Article Oxidized phospholipids protect against lung injury and endothelial barrier dysfunction caused by heat-inactivated Staphylococcus aureus.
Academic Article Alveolar Epithelial Cells Burn Fat to Survive Acute Lung Injury.
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  • Acute Lung Injury